Ambroxol is a secretolytic agent used in the treatment of respiratory diseases associated with viscid or excessive mucus. It stimulates synthesis and release of surfactant by type II pneumocytes that acts as an anti-glue factor by reducing the adhesion of mucus to the bronchial wall, in improving its transport and in providing protection against infection and irritating agents.

Ambroxol is a secretolytic agent used in the treatment of respiratory diseases associated with viscid or excessive mucus. It stimulates synthesis and release of surfactant by type II pneumocytes that acts as an anti-glue factor by reducing the adhesion of mucus to the bronchial wall, in improving its transport and in providing protection against infection and irritating agents.
Salbutamol is a beta (2)-adrenergic agonist that results in relaxation of bronchial smooth muscles by binding to beta (2)-receptors in the lungs.

A broad-spectrum anthelmintic having inhibitory effect on tubulin polymerization which results in the loss of cytoplasmic microtubules.

Levocetirizine, an active isomer of cetirizine, selectively inhibits histamine H1-receptors.

Paracetamol is an analgesic and antipyretic drug that acts primarily in the CNS, increasing the pain threshold by inhibiting cyclooxygenase, enzymes involved in prostaglandin (PG) synthesis.

Phenylephrine is a sympathomimetic amine that acts predominantly on α-adrenergic receptors working as a powerful vasoconstrictor.
A naturally occurring methylxanthine used as a pharmacological agent. Caffeine’s most notable pharmacological effect is as a central nervous system stimulant, also relaxes smooth muscle, stimulates cardiac muscle and stimulates diuresis.

A histamine H1 antagonist, Chlorpheniramine blocks the action of endogenous histamine by binding to the histamine H1 receptor, which subsequently leads to temporary relief of the negative symptoms brought on by histamine.

A potent second-generation histamine H1 antagonist, Cetirizine competes with histamine for binding at H1-receptor sites on the effector cell surface, resulting in suppression of histaminic edema, flare, and pruritus.

A second generation, tricyclic antihistamine, Desloratadine competes with free histamine for binding at H1-receptors in the GI tract, uterus, large blood vessels, and bronchial smooth muscle. This block the action of endogenous histamine, which subsequently leads to temporary relief from the negative symptoms brought on by histamine.